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Bought some Tim’s

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n***a send them my way
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Thanks this fueled me to kill myself
Refunding everything ill spend $900 on a premium rope
Nah don’t I will give you a rope for free and I can keep the tren 🤝
 
You will die
My stack is too high iq for fags like you
No your the f*g be a real man and run it without any research like I did I just popped a mystery dose of dnp
 
Yes son
Dnp isnt even that risky if you stay in the right range
Tren will give u brain damage and thats worse than dying off dnp
I’ve roided before lil bro I know
 
n***a it not even for strength
mane WHAT youre like 12 LMAOOOO ykw i believe in you
It was 6 inch or some shit idk if that’s the thick sole
w. you should try the thick doc martens too if they dont mold to your feet well. i go back & forth between the two depending on fits
 
mane WHAT youre like 12 LMAOOOO ykw i believe in you

w. you should try the thick doc martens too if they dont mold to your feet well. i go back & forth between the two depending on fits
It will make me less low dimo and then make me taller 😈😈😈
 
It will make me less low dimo and then make me taller 😈😈😈
mfk yk damn well those roids are about to close your growth plates faster than a mormon bitch close her legs
 
mfk yk damn well those roids are about to close your growth plates faster than a mormon bitch close her legs
Stanozolol has anti-estrogenic effects due to it competing for the ERa receptor with e2, meaning it prevents more e2 from affecting af2, which is the main reason for plate closure
Stanozolol actually activated the ERa af1, which causes positive bone proliferation
+ my e2 will be quite low on my stack and seeing as ER agonism from e2 is what contributes most to epiphyseal plate closure, my plate closure will be delayed
ARs arent the main reason for plate closure, which most androgens like tren target, although they can mildly affect it
ER agonism is still the main factor here
 
mane WHAT youre like 12 LMAOOOO ykw i believe in you

w. you should try the thick doc martens too if they dont mold to your feet well. i go back & forth between the two depending on fits
I don’t really like docs tbh
 
Stanozolol has anti-estrogenic effects due to it competing for the ERa receptor with e2, meaning it prevents more e2 from affecting af2, which is the main reason for plate closure
Stanozolol actually activated the ERa af1, which causes positive bone proliferation
+ my e2 will be quite low on my stack and seeing as ER agonism from e2 is what contributes most to epiphyseal plate closure, my plate closure will be delayed
ARs arent the main reason for plate closure, which most androgens like tren target, although they can mildly affect it
ER agonism is still the main factor here
n***a go eat some black beans & oats & stop playing
tho i love learning about ts so ty for the knowledge & good luck fr
just put the work in too & i 100% support (fuck roids tho)
 
n***a go eat some black beans & oats & stop playing
tho i love learning about ts so ty for the knowledge & good luck fr
just put the work in too & i 100% support (fuck roids tho)
Roids are good they dont even have any side effects
 
Roids are good they dont even have any side effects
side effects may include mogging, an amazing pump, back achne, & too many foids on your dih to even concentrate
 

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