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Deleted member 42181
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So you want to increase your DHT? Say no more fella, cause i'm about to drop another banger.. let's begin:
- Wrist size, ankle size, and bone antropometry in general is affected by DHT to an extend
- As the whole endocrine system is forming, not only DHT, but the amount of IGF-1, Gh, Testosterone/Estrogen receptors... are forming (Important for later)
- Prenatal T might be of a accurate indicator that we have high DHT
- It can increase your dick size DRAMATICALLY, talking about few more inches
- DHT extends puberty. You can have benefits of the longer puberty
- You can grow facial hair earlier than the others.
- Enlargement of larynx (Adam’s apple) and deepening of voice[3]
- Frame growth
- Higher DHT is responsible for 'logical' thinking, so you're thinking better (maturity), and sudden low inhib is result of that
- Growth of body hair, including underarm, abdominal, chest hair and pubic hair. Loss of scalp hair due to androgenic alopecia can also occur.
- You can still grow facial hair if you expose yourself to DHT later in life.
- In late puberty (17- 19), you can still deepen your voice to some extend.
- (ANECDOTAL) Some people reported growth of their frame
*Don't choose SARMS, cause they promote no androgen activity. Nandrolone derivarives don't cause balding but DHT derivarives are superior for ENCHANCING DHT specifically.
Green = Testosterone
Red = AVOID
-Taking Butea superba,which increases DHT 5X it's amount, (It doesn't cause balding so i'm pretty sure it has something to do with IGF receptors
- Taking Creatine, which supposedly increases DHT production by 50％ more itself, there are studies proving this, creatine is cheap supplement, and best you can get the most out of (Speaking of androgenmaxxing).
-Taking L-Carnitine,it sensitize the androgen receptors. Carnitine occurs naturally in meats and fish.
- Megadose Vit E, about 500mg/day, it is proven to reduce prolactin levels bu 70％, i'm going to talk about this in nofap section, just another estrogen acumulating over time causing bitch tits gynecomastia...
- Taking ashwagandha - plant that is reducing cortisol (Which further increases testosterone which is gonna be convertable to DHT later...), ashwagandha supress prolactin levels by like 15％ i recall.
- Lifting weights, hit atleast 2 bodyparts per day, and supplement magnesium after your workouts to lower cortisol. Don't go too intense because it might put your body under 'stress' mode, which will only increase cortisol and burn off additional calories - ⚠ IMPORTANT. If you want to lose weights, you are screwing your testosterone production, because like i said, you don't have the foundation to produce your hormones, even if you do something weightloss for your body:
- Starving yourself, doing high intensity workouts, sweating... It's all bad for your body and will make up bunch of cortisol because your body is on 'survival' mode methaporically speaking, and it's not looking up to preserve muscle, or hormones.
WE WANT AS LEAST CORTISOL AS POSSIBLE
* To avoid high intensity, don't do supersets between compound excercises, and give yourself respectable amount of rest
- Exposing yourself to estrogens, should i even mention this one? AVOID estrogens at all cost. Entering puberty with high estrogen = kill yourself.
- Sleep 7-9 hours, or even more, this is just an ideal range. It's best to go to sleep at 9PM because of natural melatonin production, but it's just worth mentioning right, not many people will do this. If you have trouble sleeping:
- Turn off your cellphone, or you can use it to watch some movies which will tire your eyes (Always worked for me
)
- For better sleep, supplement with:
- DHT gel, works if you are in puberty, if you aren't, idk.. give it a shot and see... Apply it on dick for pee pee gains
INTERESTING STUFF
1. This is example of person with high DHT but 0 androgen receptors, that's why you wanna make sure you take L carnitine along your hormonemax supplements, remember it activates adrogen receptors
2. Many ethnics (Middle east, north Africa, Persian area...) have high DHT production but low sensitivity to it. That's why they can grow beard earlier, and they have quite body hair. Indian subcontinent being the worst, having the lowest DHT / IGF-1 that comes with race, so they're balding, having pitch voices, short... Which totally makes DHT useless at this point to them.
3. DOCTORS DON'T KNOW ANYTHING ABOUT ENDOCRINOLOGY
- They're only learned what's dangerous about steroids, and they doesn't learn about this in college. WE KNOW MORE ABOUT THEM ON SOME FUCKING FORUM,
going to a doc is pointless and frustrating, 80% of endocrinologists/urologists dont even know that something like HCG exists, they all see injectable testosterone as a Steroid abuser Hormone, which why they only prescribe testogel, they dont even prescribe AI's
... So it seems that we can help orselves rather than relying on the help of a doctor
The effects of IGF-1 on the hair growth:
DHT BENEFITS
- IN THE WOMB
- Wrist size, ankle size, and bone antropometry in general is affected by DHT to an extend
- As the whole endocrine system is forming, not only DHT, but the amount of IGF-1, Gh, Testosterone/Estrogen receptors... are forming (Important for later)
- Prenatal T might be of a accurate indicator that we have high DHT
- IN PUBERTY
- It can increase your dick size DRAMATICALLY, talking about few more inches
- DHT extends puberty. You can have benefits of the longer puberty
- You can grow facial hair earlier than the others.
- Enlargement of larynx (Adam’s apple) and deepening of voice[3]
- Frame growth
- Higher DHT is responsible for 'logical' thinking, so you're thinking better (maturity), and sudden low inhib is result of that
- IN ADULTHDOOD
- Growth of body hair, including underarm, abdominal, chest hair and pubic hair. Loss of scalp hair due to androgenic alopecia can also occur.
- You can still grow facial hair if you expose yourself to DHT later in life.
- In late puberty (17- 19), you can still deepen your voice to some extend.
- (ANECDOTAL) Some people reported growth of their frame
HOW TO INCREASE DHT.
*Don't choose SARMS, cause they promote no androgen activity. Nandrolone derivarives don't cause balding but DHT derivarives are superior for ENCHANCING DHT specifically.
DOSAGES
Dosages depends on 50 - 250mg, remember the more the higher risk. idk which roid is better for what:
...NATURALLY?
i've mentioned things that are legit, don't ever comment 'boron' or things like 'DHEA' cause they increases estrogen and speaking of input/reward ratio, you are likely going to lose.
Blue = DHTDosages depends on 50 - 250mg, remember the more the higher risk. idk which roid is better for what:
...NATURALLY?
i've mentioned things that are legit, don't ever comment 'boron' or things like 'DHEA' cause they increases estrogen and speaking of input/reward ratio, you are likely going to lose.
Green = Testosterone
Red = AVOID
-Taking Butea superba,which increases DHT 5X it's amount, (It doesn't cause balding so i'm pretty sure it has something to do with IGF receptors
- Taking Creatine, which supposedly increases DHT production by 50％ more itself, there are studies proving this, creatine is cheap supplement, and best you can get the most out of (Speaking of androgenmaxxing).
-Taking L-Carnitine,it sensitize the androgen receptors. Carnitine occurs naturally in meats and fish.
- Megadose Vit E, about 500mg/day, it is proven to reduce prolactin levels bu 70％, i'm going to talk about this in nofap section, just another estrogen acumulating over time causing bitch tits gynecomastia...
- Taking ashwagandha - plant that is reducing cortisol (Which further increases testosterone which is gonna be convertable to DHT later...), ashwagandha supress prolactin levels by like 15％ i recall.
- Lifting weights, hit atleast 2 bodyparts per day, and supplement magnesium after your workouts to lower cortisol. Don't go too intense because it might put your body under 'stress' mode, which will only increase cortisol and burn off additional calories - ⚠ IMPORTANT. If you want to lose weights, you are screwing your testosterone production, because like i said, you don't have the foundation to produce your hormones, even if you do something weightloss for your body:
- Starving yourself, doing high intensity workouts, sweating... It's all bad for your body and will make up bunch of cortisol because your body is on 'survival' mode methaporically speaking, and it's not looking up to preserve muscle, or hormones.
DHT is more “active” after periods of rest or inaction.
(https://peweemaster7.wordpress.com/research-and-proof-dimorphism-and-ratios/)
(https://peweemaster7.wordpress.com/research-and-proof-dimorphism-and-ratios/)
WE WANT AS LEAST CORTISOL AS POSSIBLE
* To avoid high intensity, don't do supersets between compound excercises, and give yourself respectable amount of rest
- Exposing yourself to estrogens, should i even mention this one? AVOID estrogens at all cost. Entering puberty with high estrogen = kill yourself.
- Sleep 7-9 hours, or even more, this is just an ideal range. It's best to go to sleep at 9PM because of natural melatonin production, but it's just worth mentioning right, not many people will do this. If you have trouble sleeping:
- Turn off your cellphone, or you can use it to watch some movies which will tire your eyes (Always worked for me
)- For better sleep, supplement with:
- Valerian root extract
- Chamomile root extract
- Mucuna pruriens extract
- ------------------------------------------------
- DHT gel, works if you are in puberty, if you aren't, idk.. give it a shot and see... Apply it on dick for pee pee gains
STUDIES
Following baseline clinical and laboratory assessments all completed a 4-month course of daily DHT gel 2.5% (androstanolone) topically to penis (0.3 mg/kg body weight), with monitoring for adverse effects. Primary outcome was change in stretched penile length (SPL) following treatment
In the available literature, topical DHT appears to be safe when used for periods of 3–4 months at the doses noted above
Topical DHT treatment appears to be a safe and well tolerated alternative to high-dose IM testosterone for micropenis associated with PAIS. Our case series suggests that for pre- and peri-pubertal boys, this may be a very effective therapy,
sci-hub.tw/10.1515/jpem-2015-0175
Study #2
To investigate the efficacy of transdermal dihydrotestosterone therapy on 22 patients with microphallus, we applied dihydrotestosterone gel for 8 weeks to the external genitalia at daily doses of 12.5 mg. and 25 mg. for ages less than and older than 10 years, respectively
The mean increase rate (153%) in the first 4 weeks of treatment was higher than that (118%) of the second 4 weeks
Notice how even the 15year old was a good responder
sci-hub.tw/10.1016/s0022-5347(17)35576-3
In the available literature, topical DHT appears to be safe when used for periods of 3–4 months at the doses noted above
Topical DHT treatment appears to be a safe and well tolerated alternative to high-dose IM testosterone for micropenis associated with PAIS. Our case series suggests that for pre- and peri-pubertal boys, this may be a very effective therapy,
sci-hub.tw/10.1515/jpem-2015-0175
Study #2
To investigate the efficacy of transdermal dihydrotestosterone therapy on 22 patients with microphallus, we applied dihydrotestosterone gel for 8 weeks to the external genitalia at daily doses of 12.5 mg. and 25 mg. for ages less than and older than 10 years, respectively
The mean increase rate (153%) in the first 4 weeks of treatment was higher than that (118%) of the second 4 weeks
Notice how even the 15year old was a good responder
sci-hub.tw/10.1016/s0022-5347(17)35576-3
Four months of DHT treatment (50 mg im every 2 wk) in adolescent boys with delayed puberty was associated with: 1) the appearance of secondary sexual characteristics commensurate with Tanner stage II of puberty; 2) body composition changes characterized by increased lean body mass and decreased percent body fat; 3) no change in IGF-I, mean nocturnal GH, and E2 concentrations; 4) no change in rates of lipolysis; 5) no change in rates of proteolysis; 6) decreased HDL level; and 7) no change in glucose metabolism and insulin sensitivity.
Note that this dose is small compared to what i would take 50mg daily (id take 14x that dose)
after 4 months of DHT treatment, both weight and height increased (weight, 45.9 ± 3.7 vs. 49.6 ± 3.6 kg, P < 0.001; height, 149.9 ± 1.6 vs. 152.3 ± 1.5 cm, P < 0.001). Growth velocity increased from 4.4 ± 0.8 to 5.9 ± 0.9 cm/yr in five subjects in whom pretreatment growth velocity was available and to 7.1 ± 0.1 cm/yr (n = 10). Fat-free mass (FFM) increased significantly, and percentage body fat decreased
Study number 2
Significant changes in secondary sexual development were seen in both groups. Pubic hair increased from stage 1.1 + 0.1 to 2.0 f 0.5 in group A and from stage 1.2 + 0.2 to 2.2 f 0.3 in group B (P = NS). Testis size did not change appreciably in either group. Testis diameter before treatment was 5.5 + 1.1 in group A and 4.0 + 0.6 in group B (differences not significant by t test). These measurements were 5.8 f 2.2 and 4.7 f 0.8 after treatment, also not significantly different either between groups or as a result of treatment.
ted subjects grew at rates comparable to peak HV for normal pubertal boys (l), at least during the brief period of treatment. Since, in the T-treated subjects, ICGH increased at least 4-fold, and in the DHT-treated subjects, ICGH decreased almost 50%, an increase in GH secretion does not seem to be a necessary condition for androgen-stimulated growth.
sci-hub.tw/10.1210/jcem.76.4.8473416
For older subjects (keep in mind that in terms of androgenic, dht is stronger , Dihydrotestosterone has the ability to bind to sex hormone binding globulin (SHBG) more than three times higher than testosterone)
Keep in mind this used low dose of 250mg
test/3 weeks
Masculinizing therapy for F→M transsexual people was simpler in comparison, with fewer variations between patients and providers.
Note that this dose is small compared to what i would take 50mg daily (id take 14x that dose)
after 4 months of DHT treatment, both weight and height increased (weight, 45.9 ± 3.7 vs. 49.6 ± 3.6 kg, P < 0.001; height, 149.9 ± 1.6 vs. 152.3 ± 1.5 cm, P < 0.001). Growth velocity increased from 4.4 ± 0.8 to 5.9 ± 0.9 cm/yr in five subjects in whom pretreatment growth velocity was available and to 7.1 ± 0.1 cm/yr (n = 10). Fat-free mass (FFM) increased significantly, and percentage body fat decreased
Study number 2
Significant changes in secondary sexual development were seen in both groups. Pubic hair increased from stage 1.1 + 0.1 to 2.0 f 0.5 in group A and from stage 1.2 + 0.2 to 2.2 f 0.3 in group B (P = NS). Testis size did not change appreciably in either group. Testis diameter before treatment was 5.5 + 1.1 in group A and 4.0 + 0.6 in group B (differences not significant by t test). These measurements were 5.8 f 2.2 and 4.7 f 0.8 after treatment, also not significantly different either between groups or as a result of treatment.
ted subjects grew at rates comparable to peak HV for normal pubertal boys (l), at least during the brief period of treatment. Since, in the T-treated subjects, ICGH increased at least 4-fold, and in the DHT-treated subjects, ICGH decreased almost 50%, an increase in GH secretion does not seem to be a necessary condition for androgen-stimulated growth.
sci-hub.tw/10.1210/jcem.76.4.8473416
For older subjects (keep in mind that in terms of androgenic, dht is stronger , Dihydrotestosterone has the ability to bind to sex hormone binding globulin (SHBG) more than three times higher than testosterone)
Keep in mind this used low dose of 250mg
test/3 weeks
Masculinizing therapy for F→M transsexual people was simpler in comparison, with fewer variations between patients and providers.
INTERESTING STUFF
2. Many ethnics (Middle east, north Africa, Persian area...) have high DHT production but low sensitivity to it. That's why they can grow beard earlier, and they have quite body hair. Indian subcontinent being the worst, having the lowest DHT / IGF-1 that comes with race, so they're balding, having pitch voices, short... Which totally makes DHT useless at this point to them.
Mean lean muscle mass (kg)
black males = 65.6kg
White males = 62 kg
Hispanic males = 59.9 kg
Asian males = 59.6 kg
Indian males = 53.3 kg
At any given body fat mass value, South Asians had significantly less lean mass than each of the three other groups after adjustment for age, height, humerus breadth, smoking status, physical activity, and diet. Aboriginal, Chinese, and European men had 3.42 kg [95% confidence interval (CI) = 1.55–5.29], 3.01 kg (95% CI = 1.33–4.70), and 3.57 kg (95% CI = 1.82–5.33) more lean mass than South Asian men at a given total fat mass, respectively"
Ethnic Variation in Fat and Lean Body Mass and the Association with Insulin Resistance
Asian Indians tend to have more abdominal adipose tissue, less lean body mass (LBM) and higher magnitude of insulin resistance (IR) despite falling in the normal range of body mass index (BMI) [1]. The high value of waist hip ratio in Asian Indians may be due to less lean mass of the hips and greater fat at the levels of waist [2]. Another study showed that Asian Indian men have low muscle mass and 30% more total body fat (BF) than other ethnic groups [3]. Low lean mass is also evident in Asian Indian neonates as compared to white Caucasian neonates [4].
The lung capacity of Indians is 30 per cent lower than North Americans or Europeans or Chinese, making them highly vulnerable to diabetes, heart attacks or strokes, says a top scientist.
"Asian Indians had more fat, both total and in the abdominal region, with less lean mass, skeletal muscle and bone mineral than all other ethnic groups"
Body size, body composition and fat distribution: comparative analysis of European, Maori, Pacific Island and Asian Indian adults.
www.ncbi.nlm.nih.gov
"Asian Indians have different body phenotype from Europeans (36). The major differences are in high
body fat, high truncal, subcutaneous and intra-abdominal fat, and low muscle mass."
Ethnicity and type 2 diabetes in Asian Indian migrants in Auckland, New Zealand (PDF Download Available). Available from: [accessed Dec 18 2017].
"In particular, there is accumulating evidence that South Asians may have a 'low fitness' phenotype which contributes to their elevated cardio-metabolic risk, and thus may particularly benefit from undertaking higher levels of physical activity [14]"
In South Asians, a unique obesity phenotype of high abdominal fat is associated with increased cardiovascular risk
Studies in the South Asian diaspora residing in the U.K. during the early 1980s suggested the possibility of an Asian Indian or South Asian phenotype (Fig. 1). This term refers to a combination of characteristics that predisposes SA to the development of insulin resistance, type 2 diabetes, and cardiovascular disease.
black males = 65.6kg
White males = 62 kg
Hispanic males = 59.9 kg
Asian males = 59.6 kg
Indian males = 53.3 kg
At any given body fat mass value, South Asians had significantly less lean mass than each of the three other groups after adjustment for age, height, humerus breadth, smoking status, physical activity, and diet. Aboriginal, Chinese, and European men had 3.42 kg [95% confidence interval (CI) = 1.55–5.29], 3.01 kg (95% CI = 1.33–4.70), and 3.57 kg (95% CI = 1.82–5.33) more lean mass than South Asian men at a given total fat mass, respectively"
Ethnic Variation in Fat and Lean Body Mass and the Association with Insulin Resistance
Asian Indians tend to have more abdominal adipose tissue, less lean body mass (LBM) and higher magnitude of insulin resistance (IR) despite falling in the normal range of body mass index (BMI) [1]. The high value of waist hip ratio in Asian Indians may be due to less lean mass of the hips and greater fat at the levels of waist [2]. Another study showed that Asian Indian men have low muscle mass and 30% more total body fat (BF) than other ethnic groups [3]. Low lean mass is also evident in Asian Indian neonates as compared to white Caucasian neonates [4].
The lung capacity of Indians is 30 per cent lower than North Americans or Europeans or Chinese, making them highly vulnerable to diabetes, heart attacks or strokes, says a top scientist.
"Asian Indians had more fat, both total and in the abdominal region, with less lean mass, skeletal muscle and bone mineral than all other ethnic groups"
Body size, body composition and fat distribution: comparative analysis of European, Maori, Pacific Island and Asian Indian adults.
Body size, body composition and fat distribution: comparative analysis of European, Maori, Pacific Island and Asian Indian adults - PubMed
Although there is evidence that Asian Indians, Polynesians and Europeans differ in their body fat (BF)-BMI relationships, detailed comparative analysis of their underlying body composition and build characteristics is lacking. We investigated differences in the relationships between body fatness...
www.ncbi.nlm.nih.gov
"Asian Indians have different body phenotype from Europeans (36). The major differences are in high
body fat, high truncal, subcutaneous and intra-abdominal fat, and low muscle mass."
Ethnicity and type 2 diabetes in Asian Indian migrants in Auckland, New Zealand (PDF Download Available). Available from: [accessed Dec 18 2017].
"In particular, there is accumulating evidence that South Asians may have a 'low fitness' phenotype which contributes to their elevated cardio-metabolic risk, and thus may particularly benefit from undertaking higher levels of physical activity [14]"
In South Asians, a unique obesity phenotype of high abdominal fat is associated with increased cardiovascular risk
Studies in the South Asian diaspora residing in the U.K. during the early 1980s suggested the possibility of an Asian Indian or South Asian phenotype (Fig. 1). This term refers to a combination of characteristics that predisposes SA to the development of insulin resistance, type 2 diabetes, and cardiovascular disease.
3. DOCTORS DON'T KNOW ANYTHING ABOUT ENDOCRINOLOGY
- They're only learned what's dangerous about steroids, and they doesn't learn about this in college. WE KNOW MORE ABOUT THEM ON SOME FUCKING FORUM,
going to a doc is pointless and frustrating, 80% of endocrinologists/urologists dont even know that something like HCG exists, they all see injectable testosterone as a Steroid abuser Hormone, which why they only prescribe testogel, they dont even prescribe AI's
... So it seems that we can help orselves rather than relying on the help of a doctor
CO-RELATION WITH SEMEN RETENTION !?
REGARDS BALDING
Alright let's use some logic instead, cause i'm lazy to do research after all this 
. When you fap, you ejacuate some testosterone, which could've been used by your body, but you dumped that extra T...
. When you fap, you ejacuate some testosterone, which could've been used by your body, but you dumped that extra T...1. So how does our body use testosterone
- Through 5α- reduction, it either gets converted into DHT, or aromatised into estrogen. So by wasting that testosterone away, we dumped extra DHT, cause testosterone will be converted to DHT, if you aren't androgen dominant (T won't be converted to DHT), you wouldn't even have free T flowing to feel libido, motivation, energy... essentially free T is doing all the good things you think testosterone does. You can increase free T by taking proviron which lowers SHBG.
TL;DR
Masturbation = Low DHT
2. Since we know it's conversion - Does Nofap help us grow ?
- Can't answer that directly, but you saw my thread (from other forum) regarding heightmaxing, and it's discussed that DHT extends the bone growth. So by extracting that T which could be converted to DHT later, aren't we doing ourselves disadvantage in the long run?
Masturbation = Manlet
3. What happens to estrogen ?
- Meanwhile, your prolactin (Which causes low sex drive, and erectile dysfunction) is increasing. This effects in men because prolactin can stop the testes from producing testosterone. In some cases it may cause infertility, but this is pretty rare.
In males - Prolactinoma can cause decreased body or facial hair, and low bone density. Prolactin levels gets regulated to normal after one hour.
Masturbation = Prolactin release
- Through 5α- reduction, it either gets converted into DHT, or aromatised into estrogen. So by wasting that testosterone away, we dumped extra DHT, cause testosterone will be converted to DHT, if you aren't androgen dominant (T won't be converted to DHT), you wouldn't even have free T flowing to feel libido, motivation, energy... essentially free T is doing all the good things you think testosterone does. You can increase free T by taking proviron which lowers SHBG.
TL;DR
Masturbation = Low DHT
2. Since we know it's conversion - Does Nofap help us grow ?
- Can't answer that directly, but you saw my thread (from other forum) regarding heightmaxing, and it's discussed that DHT extends the bone growth. So by extracting that T which could be converted to DHT later, aren't we doing ourselves disadvantage in the long run?
Masturbation = Manlet
3. What happens to estrogen ?
- Meanwhile, your prolactin (Which causes low sex drive, and erectile dysfunction) is increasing. This effects in men because prolactin can stop the testes from producing testosterone. In some cases it may cause infertility, but this is pretty rare.
In males - Prolactinoma can cause decreased body or facial hair, and low bone density. Prolactin levels gets regulated to normal after one hour.
Masturbation = Prolactin release
"Now, on to the topic of suppressing prolactin; in this study, 300 mg’s of vitamin E for 8 weeks, decreased prolactin levels by a staggering 69% when compared to placebo in healthy human subjects."
ashwagandha:
"Furthermore, the same human study also found out that Ashwagandha lowered prolactin levels by a nice 15%."
ashwagandha:
"Furthermore, the same human study also found out that Ashwagandha lowered prolactin levels by a nice 15%."
REGARDS BALDING
The effects of IGF-1 on the hair growth:
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