Info Insight of how I grew 3,5 inches at 16 in a short time

[Graveyxrd]

I will just proceed to explain what I did and what I used etc when it comes to growing at that stage

First step is you have to acknowledge that if you’re for example 5’9 at 15 and you’re parents aren’t much taller than you or in fact shorter than you then you still have the potential to reach a extra 2-5 inches depending on many factors.

These are 2 signs that tell that you could end up taller than you actually estimate:

1). Your parents didn’t get solid nutrition or calories back in their childhood.
2). You yourself were in a calorie deficit or you were just underfed during most of your puberty.

I don’t want to get personal but I got interested in finding out that my dad and his family weren’t fed properly in that day,and I knew I could surpass this height by a lot.

I don’t want to yadayada too much so let’s get in.

Food intake
How much should you eat?
What should you eat?
When it comes to human anatomy it’s clear that you need extra resources to build more.
When you’re in a calorie deficit your body has a harder time repairing and building,the duration and effectiveness is greatly inhibited and obviously sometimes leading to catabolism.
This dosen’t only refer to muscle but also every single part of the human body and specifically the bones itself.
When you’re trying to grow but also prevent getting fat you should eat in a 10% surplus rather than anything higher.
When it comes to food sources,a big part of my journey was implementing more carbohydrates,now why?
The release of insulin has direct impact of the insulin growth factor,yet insulin sensitivity matters more and I’ll explain this later.
Also you should have 1.2g of protein per pound of body weight for optimal results.
When it comes to micronutrients,your bones are not just made out of calcium. Count in Calcium,D3,K2,Magnesium,phosphorus and zinc into it since without these supporting minerals and vitamins they wouldn’t be used as bone material and you’ll have a risk of calcification of a blood vessels etc.
Remember,you need the material to build a house and it implies with bone aswell.


50% carbohydrates | 30-35% protein | 15-20% fat

Metformin & Insulin Sensitivity
When it comes to insulin sensitivity,people underestimate the role when it comes to it and the relation it has to IGF-1.
Now I took a look at Dutch people,they have a gene that specifically enhances insulin sensitivity and they are the tallest fuckers in Europe.
First we have to look at which exact receptor IGF-1 targets and how metformin plays a role.

Now as you can see IGF-1 receptors and insulin receptors and closely related because they are part of the RTK receptors and metformin overall increases the sensitivity of every receptor.
The IGF-2 receptor is irrelevant since it’s basically only used most during pregnancy,but allowing IGF-1 bind to it’s receptor easier enhances overall growth.
1000mg between morning and afternoon and another 1000mg later in the day,with food.

Circadian Rhythm Syncing & Manipulating

This sounds stupid like the TikTok courses but what I basically did was just drink 1L of coke before I sleep with the aims of releasing and signaling IGF-1 better.
Also make sure to sleep when the sun goes down and wake up when the sun goes up,growth is bad when you’re rotting on the forum till 4 AM.
If you look at a chart on when growth hormone is released during the circadian rhythm you’ll find the highest spike at 11PM-2AM.


I hoped some of you found this thread interesting,I would drop in more things into this thread if my mind was clearer today.

 

[Currycelloser]

I will just proceed to explain what I did and what I used etc when it comes to growing at that stage

First step is you have to acknowledge that if you’re for example 5’9 at 15 and you’re parents aren’t much taller than you or in fact shorter than you then you still have the potential to reach a extra 2-5 inches depending on many factors.

These are 2 signs that tell that you could end up taller than you actually estimate:

1). Your parents didn’t get solid nutrition or calories back in their childhood.
2). You yourself were in a calorie deficit or you were just underfed during most of your puberty.

I don’t want to get personal but I got interested in finding out that my dad and his family weren’t fed properly in that day,and I knew I could surpass this height by a lot.

I don’t want to yadayada too much so let’s get in.

Food intake
How much should you eat?
What should you eat?
When it comes to human anatomy it’s clear that you need extra resources to build more.
When you’re in a calorie deficit your body has a harder time repairing and building,the duration and effectiveness is greatly inhibited and obviously sometimes leading to catabolism.
This dosen’t only refer to muscle but also every single part of the human body and specifically the bones itself.
When you’re trying to grow but also prevent getting fat you should eat in a 10% surplus rather than anything higher.
When it comes to food sources,a big part of my journey was implementing more carbohydrates,now why?
The release of insulin has direct impact of the insulin growth factor,yet insulin sensitivity matters more and I’ll explain this later.
Also you should have 1.2g of protein per pound of body weight for optimal results.
When it comes to micronutrients,your bones are not just made out of calcium. Count in Calcium,D3,K2,Magnesium,phosphorus and zinc into it since without these supporting minerals and vitamins they wouldn’t be used as bone material and you’ll have a risk of calcification of a blood vessels etc.
Remember,you need the material to build a house and it implies with bone aswell.


50% carbohydrates | 30-35% protein | 15-20% fat

Metformin & Insulin Sensitivity
When it comes to insulin sensitivity,people underestimate the role when it comes to it and the relation it has to IGF-1.
Now I took a look at Dutch people,they have a gene that specifically enhances insulin sensitivity and they are the tallest fuckers in Europe.
First we have to look at which exact receptor IGF-1 targets and how metformin plays a role.
View attachment 104243
Now as you can see IGF-1 receptors and insulin receptors and closely related because they are part of the RTK receptors and metformin overall increases the sensitivity of every receptor.
The IGF-2 receptor is irrelevant since it’s basically only used most during pregnancy,but allowing IGF-1 bind to it’s receptor easier enhances overall growth.
1000mg between morning and afternoon and another 1000mg later in the day,with food.

Circadian Rhythm Syncing & Manipulating

This sounds stupid like the TikTok courses but what I basically did was just drink 1L of coke before I sleep with the aims of releasing and signaling IGF-1 better.
Also make sure to sleep when the sun goes down and wake up when the sun goes up,growth is bad when you’re rotting on the forum till 4 AM.
If you look at a chart on when growth hormone is released during the circadian rhythm you’ll find the highest spike at 11PM-2AM.


I hoped some of you found this thread interesting,I would drop in more things into this thread if my mind was clearer today.

drinking 1 l of coke before bed is shit, u won't let ur body release growth hormone which is crucial and u should not lose on it, ur brain releases maximum growth hormone only when u sleep, so the last one is shit advice

 

[zombiebeatz2006]

Bookmarked

 

[Graveyxrd]

drinking 1 l of coke before bed is shit, u won't let ur body release growth hormone which is crucial and u should not lose on it, ur brain releases maximum growth hormone only when u sleep, so the last one is shit advice

Yes I understand what you mean with IGF-1 feedback and inhibition of growth hormone secretion.
But it all comes to the amount,and at the end IGF-1 will be the final result of growth hormone secretion,so at the end it wouldn’t matter if your pituitary gland slowed down HGH secretion because your IGF-1 which is the end product will be high

 

[Currycelloser]

Yes I understand what you mean with IGF-1 feedback and inhibition of growth hormone secretion.
But it all comes to the amount,and at the end IGF-1 will be the final result of growth hormone secretion,so at the end it wouldn’t matter if your pituitary gland slowed down HGH secretion because your IGF-1 which is the end product will be high

u can increase ur igf 1 later just not before sleep, as growth hormone is only maximally secreted during the night while u sleep whereas igf1 can be spiked at anytime so no

 

[Graveyxrd]

u can increase ur igf 1 later just not before sleep, as growth hormone is only maximally secreted during the night while u sleep whereas igf1 can be spiked at anytime so no

In order to increase IGF-1 you would need a increased growth hormone secretion at first,if your body has IGF-1 feedback that means that enough IGF-1 has been synthesized in the liver,even without a liter of coke before sleep you’d still have limited growth hormone capacity.

 

[Currycelloser]

In order to increase IGF-1 you would need a increased growth hormone secretion at first,if your body has IGF-1 feedback that means that enough IGF-1 has been synthesized in the liver,even without a liter of coke before sleep you’d still have limited growth hormone capacity.

@NewFuckingDay fact check please

 

[Graveyxrd]

@NewFuckingDay fact check please

I’m gonna honestly kill myself if I’m completely wrong
But at least what I did worked

 

[Currycelloser]

I’m gonna honestly kill myself if I’m completely wrong
But at least what I did worked

u might be partially right, I am too lazy to research abt this now but @NewFuckingDay is the goat at this so let him fact check

 

[Ray0n]

mirin

 

[NewFuckingDay]

@NewFuckingDay fact check please

u can increase ur igf 1 later just not before sleep, as growth hormone is only maximally secreted during the night while u sleep whereas igf1 can be spiked at anytime so no

This is wrong, for systemic IGF-1 to be produced you need GH (along with insulin and amino protein circulation in the blood) in order to stimulate systemic IGF-1 production, what you are thinking about is local IGF-1 secretion for instance after sprinting, which is only secreted into the muscle for the most part to aid with repair of the tissue, it doesn’t actually increase IGF-1 systemically

 

[Graveyxrd]

This is wrong, for systemic IGF-1 to be produced you need GH (along with insulin and amino protein circulation in the blood) in order to stimulate systemic IGF-1 production, what you are thinking about is local IGF-1 secretion for instance after sprinting, which is only secreted into the muscle for the most part to aid with repair of the tissue, it doesn’t actually increase IGF-1 systemically

So who’s wrong me or him

 

[NewFuckingDay]

So who’s wrong me or him

Him mostly but insulin and GH are both antagonistic to eachother which is true, but in puberty you tend to have a quite high GH release already which kind of balances it out

 

[allexxs]

I will just proceed to explain what I did and what I used etc when it comes to growing at that stage

First step is you have to acknowledge that if you’re for example 5’9 at 15 and you’re parents aren’t much taller than you or in fact shorter than you then you still have the potential to reach a extra 2-5 inches depending on many factors.

These are 2 signs that tell that you could end up taller than you actually estimate:

1). Your parents didn’t get solid nutrition or calories back in their childhood.
2). You yourself were in a calorie deficit or you were just underfed during most of your puberty.

I don’t want to get personal but I got interested in finding out that my dad and his family weren’t fed properly in that day,and I knew I could surpass this height by a lot.

I don’t want to yadayada too much so let’s get in.

Food intake
How much should you eat?
What should you eat?
When it comes to human anatomy it’s clear that you need extra resources to build more.
When you’re in a calorie deficit your body has a harder time repairing and building,the duration and effectiveness is greatly inhibited and obviously sometimes leading to catabolism.
This dosen’t only refer to muscle but also every single part of the human body and specifically the bones itself.
When you’re trying to grow but also prevent getting fat you should eat in a 10% surplus rather than anything higher.
When it comes to food sources,a big part of my journey was implementing more carbohydrates,now why?
The release of insulin has direct impact of the insulin growth factor,yet insulin sensitivity matters more and I’ll explain this later.
Also you should have 1.2g of protein per pound of body weight for optimal results.
When it comes to micronutrients,your bones are not just made out of calcium. Count in Calcium,D3,K2,Magnesium,phosphorus and zinc into it since without these supporting minerals and vitamins they wouldn’t be used as bone material and you’ll have a risk of calcification of a blood vessels etc.
Remember,you need the material to build a house and it implies with bone aswell.


50% carbohydrates | 30-35% protein | 15-20% fat

Metformin & Insulin Sensitivity
When it comes to insulin sensitivity,people underestimate the role when it comes to it and the relation it has to IGF-1.
Now I took a look at Dutch people,they have a gene that specifically enhances insulin sensitivity and they are the tallest fuckers in Europe.
First we have to look at which exact receptor IGF-1 targets and how metformin plays a role.
View attachment 104243
Now as you can see IGF-1 receptors and insulin receptors and closely related because they are part of the RTK receptors and metformin overall increases the sensitivity of every receptor.
The IGF-2 receptor is irrelevant since it’s basically only used most during pregnancy,but allowing IGF-1 bind to it’s receptor easier enhances overall growth.
1000mg between morning and afternoon and another 1000mg later in the day,with food.

Circadian Rhythm Syncing & Manipulating

This sounds stupid like the TikTok courses but what I basically did was just drink 1L of coke before I sleep with the aims of releasing and signaling IGF-1 better.
Also make sure to sleep when the sun goes down and wake up when the sun goes up,growth is bad when you’re rotting on the forum till 4 AM.
If you look at a chart on when growth hormone is released during the circadian rhythm you’ll find the highest spike at 11PM-2AM.


I hoped some of you found this thread interesting,I would drop in more things into this thread if my mind was clearer today.

nice thread

don't you think the protein is too much? I think 1.5-2g per kg is enough

wb sprints and other high intensity anaerobics? do they make a difference or is it just cope?
i heard that those if timed properly (around afternoon and early evening) actually help

 

[Graveyxrd]

nice thread

don't you think the protein is too much? I think 1.5-2g per kg is enough

wb sprints and other high intensity anaerobics? do they make a difference or is it just cope?
i heard that those if timed properly (around afternoon and early evening) actually help

The amount of protein I said was 1.2g per pound and it’s better than if you’d have the same gram amount but in kilo.
And sprints cause a short IGF-1 secretion,but this does not increase systematic serum levels in the body and you need prolonged amounts of HGH and IGF-1 for any difference to happen.
Any physical activity will spike IGF-1 secretion but also HGH secretion,but HGH is secreted because IGF-1 is being used and levels are decreasing,so at the end your total growth hormone capacity is the same and at the end those growth factors and growth hormones are used for cell repair and proliferation + hypertrophy.

 

[allexxs]

The amount of protein I said was 1.2g per pound and it’s better than if you’d have the same gram amount but in kilo.
And sprints cause a short IGF-1 secretion,but this does not increase systematic serum levels in the body and you need prolonged amounts of HGH and IGF-1 for any difference to happen.
Any physical activity will spike IGF-1 secretion but also HGH secretion,but HGH is secreted because IGF-1 is being used and levels are decreasing,so at the end your total growth hormone capacity is the same and at the end those growth factors and growth hormones are used for cell repair and proliferation + hypertrophy.

i dont really understand your logic,

physical activity spikes hgh, then hgh in the liver -> endocrine igf1
so if the sprints are timed properly with the natural hormone secretion rythm it enhances it

however autocrine igf1 can also be produced locally in the muscle tissues because of mechanical stress

autocrine igf1 does not influence hgh

hgh is not secreted because igf1 is being used, hgh is produced because of stimuli

endocrine igf1 is used for overall systemic growth (bone lenghtening,...) while autocrine igf1 is used for hypertrophy and muscle profileration.

 

[Graveyxrd]

i dont really understand your logic,

physical activity spikes hgh, then hgh in the liver -> endocrine igf1
so if the sprints are timed properly with the natural hormone secretion rythm it enhances it

however autocrine igf1 can also be produced locally in the muscle tissues because of mechanical stress

autocrine igf1 does not influence hgh

hgh is not secreted because igf1 is being used, hgh is produced because of stimuli

endocrine igf1 is used for overall systemic growth (bone lenghtening,...) while autocrine igf1 is used for hypertrophy and muscle profileration.

Fact is I don’t understand what you’re saying here.
Because physical activity spikes growth hormone to repair or proliferate damaged and healthy cells for regeneration,these growth factors and growth hormones are not used for bone lengthening but rather to adapt the body to the physical stress.
Everyone’s hormone secretion rhythm is mildly diverse between individual and other individual.
I’ve never head the term autocrine and endocrine IGF-1 since generally the IGF-1 compound is generally the same,it’s the chemical pathways that shift to send IGF-1 to the desired location the body needs.
And any kind of IGF-1 has a hormonal feedback and inhibit the amount of growth hormone released in the pituitary gland since IGF-1 is the end product of HGH in pathways with insulin and other factors.
Rather systematic and longer term IGF-1 serum levels are acquired better with the influence of insulin signaling in general.

 

[allexxs]

Fact is I don’t understand what you’re saying here.
Because physical activity spikes growth hormone to repair or proliferate damaged and healthy cells for regeneration,these growth factors and growth hormones are not used for bone lengthening but rather to adapt the body to the physical stress.
Everyone’s hormone secretion rhythm is mildly diverse between individual and other individual.
I’ve never head the term autocrine and endocrine IGF-1 since generally the IGF-1 compound is generally the same,it’s the chemical pathways that shift to send IGF-1 to the desired location the body needs.
And any kind of IGF-1 has a hormonal feedback and inhibit the amount of growth hormone released in the pituitary gland since IGF-1 is the end product of HGH in pathways with insulin and other factors.
Rather systematic and longer term IGF-1 serum levels are acquired better with the influence of insulin signaling in general.

autocrine igf1 spikes in response to mechanical stress, and is produced locally in the muscle tissues and is indeed only destined for hypertrophy and muscle profileration

endocrine igf1 is produced in the liver as it gets stimuled by hgh. hgh spikes in any high intensity aerobic movement, sleep, weights ecc.. and this one igf1 is actually used for bone lengthening and systemic overall growth, not hypertrophy and muscle repair

the point is that you previously said that somehow autocrine igf1 and hgh are in some sort of feedback loop and it is not correct, as the two are isolated and each doesnt influence the other.

endocrine igf does influence the pituitary, by reducing ghrh and gh secretion, but since it comes after the full absorption of hgh it doesnt not really make a difference

you imply that autocrine is the one that reduces hgh, because it flows im the same timespan as the hgh, but thats not true. autocrine igf does not have the same effects as endocrine igf1 on hgh levels

yes, secretion ryhtm is different from any individual to other, but by timing the high intensity anaerbics you actually manage to enhance the hormome production by producing the igf1 the moment the hgh reaches the liver in the exact 11pm-2am range

 

[Graveyxrd]

autocrine igf1 spikes in response to mechanical stress, and is produced locally in the muscle tissues and is indeed only destined for hypertrophy and muscle profileration

endocrine igf1 is produced in the liver as it gets stimuled by hgh. hgh spikes in any high intensity aerobic movement, sleep, weights ecc.. and this one igf1 is actually used for bone lengthening and systemic overall growth, not hypertrophy and muscle repair

the point is that you previously said that somehow autocrine igf1 and hgh are in some sort of feedback loop and it is not correct, as the two are isolated and each doesnt influence the other.

endocrine igf does influence the pituitary, by reducing ghrh and gh secretion, but since it comes after the full absorption of hgh it doesnt not really make a difference

you imply that autocrine is the one that reduces hgh, because it flows im the same timespan as the hgh, but thats not true. autocrine igf does not have the same effects as endocrine igf1 on hgh levels

yes, secretion ryhtm is different from any individual to other, but by timing the high intensity anaerbics you actually manage to enhance the hormome production by producing the igf1 the moment the hgh reaches the liver in the exact 11pm-2am range

@NewFuckingDay can you tell me what he’s trying to prove

 

[NewFuckingDay]

i dont really understand your logic,

physical activity spikes hgh, then hgh in the liver -> endocrine igf1
so if the sprints are timed properly with the natural hormone secretion rythm it enhances it

however autocrine igf1 can also be produced locally in the muscle tissues because of mechanical stress

autocrine igf1 does not influence hgh

hgh is not secreted because igf1 is being used, hgh is produced because of stimuli

endocrine igf1 is used for overall systemic growth (bone lenghtening,...) while autocrine igf1 is used for hypertrophy and muscle profileration.

@NewFuckingDay can you tell me what he’s trying to prove

He's trying to say physical exercise increases systemic GH levels which stimulate IGF-1 and agrees with you with the local IGF-1 exercise part, the problem is that physical exercise plays a very small role in releasing pulsatile GH at rest since the majority of our GH is produced in deep non-rem sleep anyway

 

[Graveyxrd]

He's trying to say physical exercise increases systemic GH levels which stimulate IGF-1 and agrees with you with the local IGF-1 exercise part, the problem is that physical exercise plays a very small role in releasing pulsatile GH at rest since the majority of our GH is produced in deep non-rem sleep anyway

Yeah but he randomly goes with saying “IGF-1 is endocrine and autocrine and it can be produced in the muscle magically”

 

[Deleted Member 64971]

drinking cola before sleep mogs exogenous use

 

[NewFuckingDay]

Yeah but he randomly goes with saying “IGF-1 is endocrine and autocrine and it can be produced in the muscle magically”

IGF-1 can be produced locally by muscle cells but it isn't the same as liver-derived IGF-1

 

[allexxs]

@NewFuckingDay can you tell me what he’s trying to prove

brada you were supposed to be high iq

 

[allexxs]

Yeah but he randomly goes with saying “IGF-1 is endocrine and autocrine and it can be produced in the muscle magically”

He's trying to say physical exercise increases systemic GH levels which stimulate IGF-1 and agrees with you with the local IGF-1 exercise part, the problem is that physical exercise plays a very small role in releasing pulsatile GH at rest since the majority

true but why not do it if it actually contributes even minimallly

 

[CrackyLolra]

I will just proceed to explain what I did and what I used etc when it comes to growing at that stage

First step is you have to acknowledge that if you’re for example 5’9 at 15 and you’re parents aren’t much taller than you or in fact shorter than you then you still have the potential to reach a extra 2-5 inches depending on many factors.

These are 2 signs that tell that you could end up taller than you actually estimate:

1). Your parents didn’t get solid nutrition or calories back in their childhood.
2). You yourself were in a calorie deficit or you were just underfed during most of your puberty.

I don’t want to get personal but I got interested in finding out that my dad and his family weren’t fed properly in that day,and I knew I could surpass this height by a lot.

I don’t want to yadayada too much so let’s get in.

Food intake
How much should you eat?
What should you eat?
When it comes to human anatomy it’s clear that you need extra resources to build more.
When you’re in a calorie deficit your body has a harder time repairing and building,the duration and effectiveness is greatly inhibited and obviously sometimes leading to catabolism.
This dosen’t only refer to muscle but also every single part of the human body and specifically the bones itself.
When you’re trying to grow but also prevent getting fat you should eat in a 10% surplus rather than anything higher.
When it comes to food sources,a big part of my journey was implementing more carbohydrates,now why?
The release of insulin has direct impact of the insulin growth factor,yet insulin sensitivity matters more and I’ll explain this later.
Also you should have 1.2g of protein per pound of body weight for optimal results.
When it comes to micronutrients,your bones are not just made out of calcium. Count in Calcium,D3,K2,Magnesium,phosphorus and zinc into it since without these supporting minerals and vitamins they wouldn’t be used as bone material and you’ll have a risk of calcification of a blood vessels etc.
Remember,you need the material to build a house and it implies with bone aswell.


50% carbohydrates | 30-35% protein | 15-20% fat

Metformin & Insulin Sensitivity
When it comes to insulin sensitivity,people underestimate the role when it comes to it and the relation it has to IGF-1.
Now I took a look at Dutch people,they have a gene that specifically enhances insulin sensitivity and they are the tallest fuckers in Europe.
First we have to look at which exact receptor IGF-1 targets and how metformin plays a role.
View attachment 104243
Now as you can see IGF-1 receptors and insulin receptors and closely related because they are part of the RTK receptors and metformin overall increases the sensitivity of every receptor.
The IGF-2 receptor is irrelevant since it’s basically only used most during pregnancy,but allowing IGF-1 bind to it’s receptor easier enhances overall growth.
1000mg between morning and afternoon and another 1000mg later in the day,with food.

Circadian Rhythm Syncing & Manipulating

This sounds stupid like the TikTok courses but what I basically did was just drink 1L of coke before I sleep with the aims of releasing and signaling IGF-1 better.
Also make sure to sleep when the sun goes down and wake up when the sun goes up,growth is bad when you’re rotting on the forum till 4 AM.
If you look at a chart on when growth hormone is released during the circadian rhythm you’ll find the highest spike at 11PM-2AM.


I hoped some of you found this thread interesting,I would drop in more things into this thread if my mind was clearer today.

W post. More people need to know about insulin factors

 

[Graveyxrd]

brada you were supposed to be high iq

There’s a difference between IGF-1 synthesis and IGF-1 secretion but endocrine and autocrine IGF-1 sounds like bullshit to me.
Also It wouldn’t make sense logically since why would there be a form IGF-1 that specifically increase bone length,it’s common knowledge that depending on bodily and cellular need IGF-1 is sent to the desired cells which need it and there’s clearly only one version of IGF-1 and this is it:

I could be a bit wrong since I might not totally understand what you mean with endocrine and autocrine IGF-1.

 

[allexxs]

There’s a difference between IGF-1 synthesis and IGF-1 secretion but endocrine and autocrine IGF-1 sounds like bullshit to me.
Also It wouldn’t make sense logically since why would there be a form IGF-1 that specifically increase bone length,it’s common knowledge that depending on bodily and cellular need IGF-1 is sent to the desired cells which need it and there’s clearly only one version of IGF-1 and this is it:
View attachment 104543
I could be a bit wrong since I might not totally understand what you mean with endocrine and autocrine IGF-1.

i know what you mean it sounded illogic to me too but there are plenty of studies supporing the idea of there being autocrine and endocrine igf-1

the logic is that autocrine is made by the cells and muslce tissues and stays there and so is only used for hypertrophy and muscle recovery

it only acts locally

endocrine igf1 instead enters the entire bloodstream and affects the whole body

 

[Graveyxrd]

i know what you mean it sounded illogic to me too but there are plenty of studies supporing the idea of there being autocrine and endocrine igf-1

the logic is that autocrine is made by the cells and muslce tissues and stays there and so is only used for hypertrophy and muscle recovery

it only acts locally

endocrine igf1 instead enters the entire bloodstream and affects the whole body

I get what you mean with this theory but this seems a bit off.
Simply because you need GH to synthesize IGF-1 since it’s specifically synthesized in the liver.
The whole point of GH is to derive and synthesize IGF-1 and other growth factors and no other place in the body except the liver is this possible.
Also I talked about chemical pathways and cellular requesting,wherever the body needs IGF-1 it sends it to anywhere.
For example,after a workout cells get damaged and in need for repair,IGF-1 is secreted into the muscle by bloodstream to reach those cells.
I’m sorry but I seriously can’t wrap my mind around different forms of IGF-1 and you should really look carefully at it’s biochemical purposes and journey.

 

[Decay]

Sleeping at 4AM doesn’t stunt much growth if any at all if you’re still getting 8-10 hours (although sleeping at a more appropriate time is better for overall health) good guide though.

 

[Graveyxrd]

Sleeping at 4AM doesn’t stunt much growth if any at all if you’re still getting 8-10 hours (although sleeping at a more appropriate time is better for overall health) good guide though.

You’d still get sleep but that sleep is worse no matter the duration than sleep synched with the circadian rhythm

 

[allexxs]

I get what you mean with this theory but this seems a bit off.
Simply because you need GH to synthesize IGF-1 since it’s specifically synthesized in the liver.
The whole point of GH is to derive and synthesize IGF-1 and other growth factors and no other place in the body except the liver is this possible.
Also I talked about chemical pathways and cellular requesting,wherever the body needs IGF-1 it sends it to anywhere.
For example,after a workout cells get damaged and in need for repair,IGF-1 is secreted into the muscle by bloodstream to reach those cells.
I’m sorry but I seriously can’t wrap my mind around different forms of IGF-1 and you should really look carefully at it’s biochemical purposes and journey.

gh stimulates the synthesis of igf-1, it doesnt actually do it itself

 

[allexxs]

Sleeping at 4AM doesn’t stunt much growth if any at all if you’re still getting 8-10 hours (although sleeping at a more appropriate time is better for overall health) good guide though.

circadian rythm is actually a thing if you sleep at 4 am i think its way worse

 

[NewFuckingDay]

true but why not do it if it actually contributes even minimallly

Because it is like a major waste of time for just less than a IU equivalent of GH, just exercise normally as you would

 

[allexxs]

Because it is like a major waste of time for just less than a IU equivalent of GH, just exercise normally as you would

Combined sprint and resistance training abrogates age differences in somatotropic hormones - PMC

The aim of this investigation was to compare serum growth hormone (GH), insulin-like growth factor-1 (IGF-1) and insulin-like growth factor-binding protein-3 (IGFBP-3) in response to a combined sprint and resistance training (CSRT) program in young ...

pmc.ncbi.nlm.nih.gov

its actually a 5-15% difference

 

[NewFuckingDay]

Combined sprint and resistance training abrogates age differences in somatotropic hormones - PMC

The aim of this investigation was to compare serum growth hormone (GH), insulin-like growth factor-1 (IGF-1) and insulin-like growth factor-binding protein-3 (IGFBP-3) in response to a combined sprint and resistance training (CSRT) program in young ...

pmc.ncbi.nlm.nih.gov

its actually a 5-15% difference

That's literally nothing bro, pretty sure taking a tab of L-arginine has a larger increase than that

 

[allexxs]

That's literally nothing bro, pretty sure taking a tab of L-arginine has a larger increase than

nah l arginine has a smaller increase on gh at rest than sprinting

 

[Currycelloser]

nah l arginine has a smaller increase on gh at rest than sprinting

Gh increase after sprints is useless

 

[allexxs]

Gh increase after sprints is useless

Combined sprint and resistance training abrogates age differences in somatotropic hormones - PMC

The aim of this investigation was to compare serum growth hormone (GH), insulin-like growth factor-1 (IGF-1) and insulin-like growth factor-binding protein-3 (IGFBP-3) in response to a combined sprint and resistance training (CSRT) program in young ...

pmc.ncbi.nlm.nih.gov

 

[Currycelloser]

Combined sprint and resistance training abrogates age differences in somatotropic hormones - PMC

The aim of this investigation was to compare serum growth hormone (GH), insulin-like growth factor-1 (IGF-1) and insulin-like growth factor-binding protein-3 (IGFBP-3) in response to a combined sprint and resistance training (CSRT) program in young ...

pmc.ncbi.nlm.nih.gov

What I meant is that the gh released after sprints won’t contribute to height growth

 

[Graveyxrd]

Combined sprint and resistance training abrogates age differences in somatotropic hormones - PMC

The aim of this investigation was to compare serum growth hormone (GH), insulin-like growth factor-1 (IGF-1) and insulin-like growth factor-binding protein-3 (IGFBP-3) in response to a combined sprint and resistance training (CSRT) program in young ...

pmc.ncbi.nlm.nih.gov

its actually a 5-15% difference

The elevation of growth hormones will literally not contribute to bone growth.
Any physical stress will cause temporary microdamage in the body as you know,not only muscle but other tissue and I already explained why it wouldn’t contribute to growth is any bone due to cellular demand.
At the end,hormone levels don’t matter unless it’s actually used for desired goals.
When you work out also other hormones such as androgenic ones are also released but are used for repair and adaptation rather than proliferation.

 

[ElvayneMogs]

So who’s wrong me or him

I will just proceed to explain what I did and what I used etc when it comes to growing at that stage

First step is you have to acknowledge that if you’re for example 5’9 at 15 and you’re parents aren’t much taller than you or in fact shorter than you then you still have the potential to reach a extra 2-5 inches depending on many factors.

These are 2 signs that tell that you could end up taller than you actually estimate:

1). Your parents didn’t get solid nutrition or calories back in their childhood.
2). You yourself were in a calorie deficit or you were just underfed during most of your puberty.

I don’t want to get personal but I got interested in finding out that my dad and his family weren’t fed properly in that day,and I knew I could surpass this height by a lot.

I don’t want to yadayada too much so let’s get in.

Food intake
How much should you eat?
What should you eat?
When it comes to human anatomy it’s clear that you need extra resources to build more.
When you’re in a calorie deficit your body has a harder time repairing and building,the duration and effectiveness is greatly inhibited and obviously sometimes leading to catabolism.
This dosen’t only refer to muscle but also every single part of the human body and specifically the bones itself.
When you’re trying to grow but also prevent getting fat you should eat in a 10% surplus rather than anything higher.
When it comes to food sources,a big part of my journey was implementing more carbohydrates,now why?
The release of insulin has direct impact of the insulin growth factor,yet insulin sensitivity matters more and I’ll explain this later.
Also you should have 1.2g of protein per pound of body weight for optimal results.
When it comes to micronutrients,your bones are not just made out of calcium. Count in Calcium,D3,K2,Magnesium,phosphorus and zinc into it since without these supporting minerals and vitamins they wouldn’t be used as bone material and you’ll have a risk of calcification of a blood vessels etc.
Remember,you need the material to build a house and it implies with bone aswell.


50% carbohydrates | 30-35% protein | 15-20% fat

Metformin & Insulin Sensitivity
When it comes to insulin sensitivity,people underestimate the role when it comes to it and the relation it has to IGF-1.
Now I took a look at Dutch people,they have a gene that specifically enhances insulin sensitivity and they are the tallest fuckers in Europe.
First we have to look at which exact receptor IGF-1 targets and how metformin plays a role.
View attachment 104243
Now as you can see IGF-1 receptors and insulin receptors and closely related because they are part of the RTK receptors and metformin overall increases the sensitivity of every receptor.
The IGF-2 receptor is irrelevant since it’s basically only used most during pregnancy,but allowing IGF-1 bind to it’s receptor easier enhances overall growth.
1000mg between morning and afternoon and another 1000mg later in the day,with food.

Circadian Rhythm Syncing & Manipulating

This sounds stupid like the TikTok courses but what I basically did was just drink 1L of coke before I sleep with the aims of releasing and signaling IGF-1 better.
Also make sure to sleep when the sun goes down and wake up when the sun goes up,growth is bad when you’re rotting on the forum till 4 AM.
If you look at a chart on when growth hormone is released during the circadian rhythm you’ll find the highest spike at 11PM-2AM.


I hoped some of you found this thread interesting,I would drop in more things into this thread if my mind was clearer today.

Yo , I am 172 cm and 63kg at 15 , my mom is 152 but my dad is 182 so do you think with a good sleep schedule and a good alimentation I could maybe reach 180 ?

 

[Graveyxrd]

Yo , I am 172 cm and 63kg at 15 , my mom is 152 but my dad is 182 so do you think with a good sleep schedule and a good alimentation I could maybe reach 180 ?

Definitely,there’s still so much time.
For best results you could follow everything I stated,it dosen’t necessarily have to be metformin but any glucagon agonist that you can get your hands on.

 

[ElvayneMogs]

Definitely,there’s still so much time.
For best results you could follow everything I stated,it dosen’t necessarily have to be metformin but any glucagon agonist that you can get your hands on.

May I mp you ? because I think I don't understand everything u said before

 

[Graveyxrd]

May I mp you ? because I think I don't understand everything u said before

Sure