DerangedAutto
New member
- Joined
- Oct 26, 2021
- Messages
- 3
- Reputation
- 5
Alright, folks, listen up,
We all know size matters, and anyone who says otherwise is coping.
It seems as if there are little to no solutions on the internet to this epidemic of dickletism, like on one hand you have delusional bluepillers on Reddit saying things like, “The g-spot is only 3 inches deep, 5.5 inches is more than enough!” and on the other side of the spectrum it isn’t much better either, you have people telling you to lynch your fucking cock like a powerlifter in hopes that it will elongate it, yeah no thanks!
I don’t you about you guys, but deluding myself ending up with a shitty sex life or torturing my cock into nerve damage and ED just doesn’t seem ideal to me. There’s gotta be another way.
The problem I see with the version of PE we’re most familiar with (Jelqing, pumping, hanging, extender devices, etc.) is that the options that we’re given come with trade-offs, those being nerve damage, busting blood vessels, scar tissue, and just a loss of functionality in general. Who tf wants a big dick at the cost of all of those damages? I know I don’t, and I can certainly doubt that you don’t either.
Btw don’t tell us to use Viagra/Cialis, we’re not old dudes with limp dicks, and using such compounds isn’t real PE, that’s just reaching your potential, we’re set on extending our potential.
Alright, now let’s talk about potential solutions. What I believe the best solution to be is chemical PE, nothing that you haven’t heard of already. Now I want to make sure everyone is on the same page as me, when I say chemical PE, I’m thinking about manipulating the body into inducing growth in the corpus cavernosal/spongiosum, so NOT allowing more blood flow. Guys with bigger dicks have more erectile tissue, and that’s what we ourselves are trying to get more of through a means of pharmacological usage.
Now I’m not an endocrinologist, so there’s a lot I don’t know about hormones and anatomy, so allow me to ask a few questions to point us in the right direction:
-How does the penis stop growing, not when, but how?
-Assuming that it stops growing by a loss in sensitivity to DHT, is that sensitivity lost through a means of time since it’s last been exposed to DHT, or is that sensitivity lost due to the amount of DHT?
-What differentiates guys with average dicks from guys with big dicks hormone-wise?
-Is dick size determined by DHT capacity or amount of DHT exposure during puberty?
-I’ve heard theories about how taking finasteride (Stops DHT production in the body) for long periods of time will resensitize your dick to DHT and open up a window of opportunity for dick growth, is this true?
-With that said, will finasteride shrink your dick if attempting this?
Ok, now that I’ve gotten that out of the way, I just want to say that we should just put our minds together and go on a quest for finding an effective and permanent solution. Let’s be supportive of this and work together.
We all know size matters, and anyone who says otherwise is coping.
It seems as if there are little to no solutions on the internet to this epidemic of dickletism, like on one hand you have delusional bluepillers on Reddit saying things like, “The g-spot is only 3 inches deep, 5.5 inches is more than enough!” and on the other side of the spectrum it isn’t much better either, you have people telling you to lynch your fucking cock like a powerlifter in hopes that it will elongate it, yeah no thanks!
I don’t you about you guys, but deluding myself ending up with a shitty sex life or torturing my cock into nerve damage and ED just doesn’t seem ideal to me. There’s gotta be another way.
The problem I see with the version of PE we’re most familiar with (Jelqing, pumping, hanging, extender devices, etc.) is that the options that we’re given come with trade-offs, those being nerve damage, busting blood vessels, scar tissue, and just a loss of functionality in general. Who tf wants a big dick at the cost of all of those damages? I know I don’t, and I can certainly doubt that you don’t either.
Btw don’t tell us to use Viagra/Cialis, we’re not old dudes with limp dicks, and using such compounds isn’t real PE, that’s just reaching your potential, we’re set on extending our potential.
Alright, now let’s talk about potential solutions. What I believe the best solution to be is chemical PE, nothing that you haven’t heard of already. Now I want to make sure everyone is on the same page as me, when I say chemical PE, I’m thinking about manipulating the body into inducing growth in the corpus cavernosal/spongiosum, so NOT allowing more blood flow. Guys with bigger dicks have more erectile tissue, and that’s what we ourselves are trying to get more of through a means of pharmacological usage.
Now I’m not an endocrinologist, so there’s a lot I don’t know about hormones and anatomy, so allow me to ask a few questions to point us in the right direction:
-How does the penis stop growing, not when, but how?
-Assuming that it stops growing by a loss in sensitivity to DHT, is that sensitivity lost through a means of time since it’s last been exposed to DHT, or is that sensitivity lost due to the amount of DHT?
-What differentiates guys with average dicks from guys with big dicks hormone-wise?
-Is dick size determined by DHT capacity or amount of DHT exposure during puberty?
-I’ve heard theories about how taking finasteride (Stops DHT production in the body) for long periods of time will resensitize your dick to DHT and open up a window of opportunity for dick growth, is this true?
-With that said, will finasteride shrink your dick if attempting this?
Ok, now that I’ve gotten that out of the way, I just want to say that we should just put our minds together and go on a quest for finding an effective and permanent solution. Let’s be supportive of this and work together.
