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Info Why “Good Skin” Isn’t Skincare — It’s Low Inflammation

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I keep seeing people on here obsess over routines, actives, percentages, and product stacks. From a medical point of view, that’s missing the actual mechanism.

What people read as “good skin” isn’t smoothness or oil control. It’s physiological stability. Your skin is basically a billboard advertising how stressed your body is.

You can have average bone structure and still look high tier if your skin signals low systemic stress.

Here’s what I mean.

Most skin problems aren’t caused by a lack of products — they’re caused by chronic low-grade inflammation that never quite crosses into disease. Not acne you can treat with antibiotics. Not eczema you can diagnose. Just constant background irritation.

The biggest contributors aren’t talked about enough: – sleep debt
– blood sugar swings
– over-washing
– psychological stress
– and yes, too many actives

Cortisol alone suppresses barrier repair. When the barrier weakens, your skin loses water faster, nerve endings fire more, cytokines increase, and you get that dull, reddish, uneven look people mistake for “bad genetics.”

This is why some guys run water + moisturizer and look insane, while others do ten steps and stay inflamed.

Over-skincare is a real thing. Dermatology calls it irritant dermatitis. Looksmaxxing calls it “why does my face always look tired and red even though I do everything right?”

Another unpopular truth: sebum isn’t the enemy. Oxidized sebum is.

Sebum is protective. It’s antioxidant. It’s antimicrobial. The problem is UV, pollution, smoking, and aggressive cleansing stripping away its protective components. Dry skin doesn’t read as clean — it reads as stressed.

Balanced oil reflects light better than matte skin ever will.

That’s also why people underestimate circulation. Skin with good microvascular flow reflects light evenly. Skin with poor flow looks flat no matter how clear it is.

Exercise, heat exposure, even simple facial massage increases nitric oxide locally. That “glow” people chase with products is often just blood flow.

Tone matters more than color. Even skin > pale skin > tan skin. Patchy redness, uneven melanin, and dullness signal inflammation subconsciously. Humans are good at spotting it even if they don’t know why.

The gut-skin stuff is mostly noise, except for one real factor: endotoxemia. Ultra-processed diets increase intestinal permeability. Lipopolysaccharides leak into circulation. Skin flares. That’s it. That’s the mechanism.

Some people fix their skin with diet overnight because they actually had that problem. Others don’t see changes because they didn’t.

The real looksmaxxing play isn’t adding more. It’s subtracting inflammation.

Stop nuking your face. Stop chasing dryness. Fix sleep. Stabilize blood sugar. Let the barrier recover.

Once your skin stops being irritated, it does the rest on its own.

That’s the difference between cosmetic skincare and medical skin optimization.
 
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The biggest contributors aren’t talked about enough: – sleep debt
– blood sugar swings
– over-washing
– psychological stress
– and yes, too many actives
true dnr everything else lots of other factors js pin ghk-cu and forget abt acne
 
blood sugar swings
basically attached to your sebaceous glands are specialized cells called sebocytes, these produce sebum, along the cell membrane are hormone receptors these cells express androgen receptors (testosterone and DHT) and are responsive to IGF-1. High-glycaemic diets increase insulin, which raises bioavailable IGF-1. IGF-1 and androgens together enhance sebocytes proliferation and sebum production, while DHT, being the most potent androgen, strongly stimulates sebaceous activity. Excess sebum, combined with follicular blockage and inflammation, contributes to acne development.
 

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